BRUCE EFFECT COMPETENCE IN YELLOW-LETHAL HETEROZYGOUS MICE
نویسندگان
چکیده
منابع مشابه
Bruce effect competence in yellow-lethal heterozygous mice.
The capacity of heterozygous yellow-lethal (Aya) and brown (aa) male mice to induce the Bruce effect was investigated. The proportion of SJL/J females showing a failure of implantation when exposed to males of either genotype was 80 %, whereas only 20 % of the control females not so exposed showed pregnancy failure. One-half of the females whose pregnancy had been blocked as a result of exposur...
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in the present study, the effect of e-64 at different concentrations (0.5, 1, 5 and 10 µm) added to (1) the ivm medium on oocyte nuclear maturation and developmental competence of ovine oocytes, and (2) to the ivc medium on embryonic development of ovine embryos were investigated.
Heterozygous Mice
A number of alleles at coat color loci of the house mouse give rise to areas of wild-type pigmentation on the coats of otherwise mutant animals. Such unstable alleles include both recessive and dominant mutations. Among the latter are several alleles at the W locus. In this report, phenotypic reversions of the W allele at the W locus were studied Mice heterozygous in repulsion for both We' and ...
متن کاملTransient receptor potential melastatin 6 knockout mice are lethal whereas heterozygous deletion results in mild hypomagnesemia.
BACKGROUND Hypomagnesemia with secondary hypocalcemia is due to disturbed renal and intestinal magnesium (Mg(2+)) (re)absorption. The underlying defect is a mutation in the transient receptor potential melastatin type 6 (TRPM6), a Mg(2+)-permeable ion channel expressed in the kidney and intestine. Our aim was to characterize homozygous (-/-) and heterozygous (+/-) TRPM6 knockout mice with respe...
متن کاملTransient Receptor Potential Melastatin 6 Knockout Mice Are Lethal whereas Heterozygous Deletion Results in Mild Hypomagnesemia
Background: Hypomagnesemia with secondary hypocalcemia is due to disturbed renal and intestinal magnesium (Mg 2+ ) (re)absorption. The underlying defect is a mutation in the transient receptor potential melastatin type 6 (TRPM6), a Mg 2+ -permeable ion channel expressed in the kidney and intestine. Our aim was to characterize homozygous (–/–) and heterozygous (+/–) TRPM6 knockout mice with resp...
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ژورنال
عنوان ژورنال: Reproduction
سال: 1974
ISSN: 1470-1626,1741-7899
DOI: 10.1530/jrf.0.0400483